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Tactile agnosia case study

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Two patients with tactile naming disorders are reported. Case 1 right hand tactile agnosia due to bilateral cerebral infarction differentiated tactile qualities of objects normally, but could neither name nor categorize the objects. Case 2 bilateral tactile aphasia after operation of an epidural left parietal haematoma had as severe a tactile naming disturbance as Case 1, but could categorize objects normally, demonstrating that tactile recognition was preserved. Case 1 may be the first case of tactile agnosia clearly differentiated from tactile aphasia. CT scans of Case 1 revealed lesions in the left angular gyrus, and in the right parietal, temporal, and occipital lobes. Case 2 had lesions in the left angular gyrus and of posterior callosal radiations.

The nature of tactile agnosia: A case study

Tactile agnosia case study
Tactile agnosia case study
Tactile agnosia case study

Central touch disorders - Scholarpedia

Reed and Richard J. Abstract-A chronic tactile agnosic with a small, MRI-documented left inferior parietal infarction underwent detailed somesthetic testing to assess 1 the acquisition of sensory data, 2 the manipulation of somatosensory percept and its association with previous knowledge, and 3 recognition occurring at a deeper taxonomic level. Results suggest that tactile agnosia can arise from faulty high-level perceptual processes, but that the ability to associate tactually defined objects and object parts with episodic memory can be preserved. Consistent with anatomic and physiologic studies in nonhuman primates, inferior parietal cortex including Brodmann area 40, possibly area 39 appears to serve as a high-level somatosensory region. Documents: Advanced Search Include Citations. Authors: Advanced Search Include Citations. Reed , Richard J.

A case of tactile agnosia with a lesion restricted to the post-central gyrus

Catherine L. Caselli , Mayo Clinic. A chronic tactile agnosic with a small, MRI-documented left inferior parietal infarction underwent detailed somesthetic testing to assess 1 the acquisition of sensory data, 2 the manipulation of somatosensory percept and its association with previous knowledge, and 3 recognition occurring at a deeper taxonomic level. Results suggest that tactile agnosia can arise from faulty high-level perceptual processes, but that the ability to associate tactually defined objects and object parts with episodic memory can be preserved.
Click on image for details. Tactile agnosia has been described after lesions of the primary sensory cortex but the exact location and extension of those lesions is not clear. We report the clinical features and imaging findings in a patient with an acute ischemic stroke restricted to the primary sensory area S1. A year-old man had a sudden onset of a left alien hand, without left hemiparesis. Neurological examination showed intact primary sensory functions, but impaired recognition of shape, size macrogeometrical and texture microgeometrical of objects; damage confined to the post-central gyrus, sparing the posterior parietal cortex was demonstrated on MRI.
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